#AltriNon

Tomorrow is the day!
From Tuesday 21st to Friday 24th, Santiago de Compostela will be the capital city of the Spanish Cell Biology community.
We will welcome close to 200 attendees that will share their knowledge. looking forward!!

hahahah <3

So happy for you @frisophagy.bsky.social!! Very well deserved! many years of hard work finally have a great pay off! Congratulations 😍

Tomorrow last day for early registation. Do not miss the opportunity and join us!!!

Thanks to you for sharing with us your science and friendship! Long life to @mitowomen.bsky.social #femaleinscience 💪🏻💪🏻💪🏻. Next stop Santiago de Compostela at our SEBC meeting in October!🤗🤗🤗

What a presentation @jorgms.bsky.social! You blew oir minds! So happy you joined us!! 💕💕🤗

Home after a fantatastic week in Cáceres for the 47th SEBBM meeting! Thank you very much @sabiolab.bsky.social @pgonzalezrodriguez.bsky.social @juanaloca2002.bsky.social and the rest of the organisers for the hard work and the patience! You guys rock! SEBBM is the life example of #womeninscience!💪🏻💪🏻

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Very proud of my friend Diana Guallar @epiageing-lab.bsky.social who has been awarded with the ‘Mujer y Ciencia Award” this year by SEBBM at the #47thSEBBMmeeting! For her work carried at @cimususc.bsky.social

Last days for earlybird registration! Lots of travel grants for students and postdocs and many opportunities for short talks! Join us in Santiago de Compostela!

Proud to see our Rafael Garrido Fernandez presenting his data at SEBBM 47th meeting. If you are interested in knowing more about mtDNA signaling in LUAD! Go and visit Rafa at póster 288 next thursday and friday.

What an honor veinte at the cams this year! Thanks a lot @fafianlabora.bsky.social for having us!!!

SUMOylation of the lysine-less tumor suppressor p14ARF counters ubiquitylation-dependent degradation Cell Death & Disease - SUMOylation of the lysine-less tumor suppressor p14ARF counters ubiquitylation-dependent degradation

Happy to see this fantastic paper by @mela-virus.bsky.social and her lab at @cimususc.bsky.social in which we have collaborated, finally published in Cell Death & Disease, “SUMOylation of the lysine-less tumor suppressor p14ARF counters ubiquitylation-dependent degradation”:
rdcu.be/evZx9

This is figure 1, which shows DDHD2 lipase localizes to synaptic terminals in the hippocampus.

A study in Nature Metabolism shows that under glucose-depleted conditions, neurons can use fatty acids as an alternative source of energy to support synaptic function. go.nature.com/44T8kct 🧪

Don’t Miss the Spanish Society of Cell Biology (SEBC) Meeting 2025!
📍 Santiago de Compostela
📅 October 21–24, 2025

👉 Register now at sebc2025.org to:

Secure your discounted registration

Apply for fellowships covering the registration fee (available for junior researchers!)

Happy to see many (and mito) spanish scientists in the awardees of the Advanced Grant 2024! Special congratulations @genoxphos.bsky.social,
Ana Garcia Saez, @bolanosjuanp.bsky.social as well to all the others! erc.europa.eu/sites/defaul...

Inherited mitochondrial genetics as a predictor of immune checkpoint inhibition efficacy in melanoma - Nature Medicine In this genomic analysis of peripheral blood samples of the phase 3 CheckMate-067 trial of ipilimumab (IPI) versus nivolumab (NIVO) versus ipilimumab and nivolumab (IPI-NIVO) in melanoma, the status o...

Inherited mitochondrial genetics as a
predictor of immune checkpoint inhibition
efficacy in melanoma www.nature.com/articles/s41... and many other cancer types and therapies! Mind the mtDNA population genetics please!

Mindblowing!!!

Nuestro grupo oferta una de ellos!! Venir a disfrutar de la ciencia mitocondrial con nosotros!!! ❤️❤️

Do not miss our webinar tomorrow with 🗣️ @tabaralc.bsky.social and
🗣️ @dplamartin.bsky.social lots of mitochondrial fun!!

you guys look fantastic!

Great to be working alongside @mitocamb.bsky.social @ucl-qs-cnmd.bsky.social @lilyfoundation.bsky.social and @mitonewcastle.bsky.social colleagues in this new Centre!

Don´t miss the opportunity to be part of this fantastic meeting with a stellar lineup of speakers, loads of opportunities to present your work, and in beautiful Santiago de Compostela!!!! Sign up!!

Running a genetic stop sign accelerates oxygen metabolism and energy production in horses Horses are among nature’s greatest athletes, yet the ancestral molecular adaptations fueling their energy demands are poorly understood. Within a clinically important pathway regulating redox and meta...

Running a genetic stop sign accelerates oxygen metabolism and energy production in horses

Mind-blowing talk today by @martakova.bsky.social today on metabolic regulation of plasticity and beyond!! Watch this space!

In-cell architecture of the mitochondrial respiratory chain Mitochondria regenerate adenosine triphosphate (ATP) through oxidative phosphorylation. This process is carried out by five membrane-bound complexes collectively known as the respiratory chain, workin...

In-cell architecture of the mitochondrial respiratory chain | Science www.science.org/doi/10.1126/...

The time is now: accounting for time-of-day effects to improve reproducibility and translation of metabolism research - Nature Metabolism This broad group of authors summarizes the impact of circadian factors on metabolic biology and offers recommendations on how to account for and report biological, environmental and experimental facto...

The importance of considering time of day for metabolic experiments

www.nature.com/articles/s42...

Engineering mtDNA deletions by reconstituting end joining in human mitochondria Recent breakthroughs in the genetic manipulation of mitochondrial DNA (mtDNA) have enabled precise base substitutions and the efficient elimination of…

Combining bacterial end joining with programmable nucleases, we induced mtDNA deletions in human cells at varying heteroplasmy and examined their metabolic and cellular effects. Simple tool to apply across any cell type and engineer pathogenic mtDNA deletions. www.sciencedirect.com/science/arti...

Engineering mtDNA deletions by reconstituting end joining in human mitochondria Combined expression of a mitochondrially targeted endonuclease and end-joining machinery enables formation of mtDNA deletions with a broad range of heteroplasmy.

Engineering mtDNA deletions by reconstituting end joining in human mitochondria: Cell www.cell.com/cell/fulltex...

Cryptic mitochondrial DNA mutations coincide with mid-late life and are pathophysiologically informative in single cells across tissues and species - Nature Communications Mitochondrial dysfunction is implicated in ageing. Here the authors report that across tissues and species, somatic mitochondrial DNA mutations in single cells (termed cryptic mutations) accumulate wi...

Cryptic mitochondrial DNA mutations coincide with mid-late life and are pathophysiologically informative in single cells across tissues and species
www.nature.com/articles/s41...