Company Updates: Spring 2026 | StressMarq StressMarq’s product development is driven by an ongoing dialogue between our scientists & the research community. Learn about our new products for Spring 2026.

🌱 StressMarq is excited to welcome spring with a fresh lineup of #AlphaSynuclein, #Tau, and #AmyloidBeta products.

🔎 Discover our approach to product innovation, upcoming conferences, and our ongoing commitment to advancing #neurodegeneration research worldwide 🌸 https://bit.ly/40VPtLf

StressMarq will be exhibiting at #ADPD2026 from March 17-21 in Copenhagen!📍

Visit us at Booth No. 70 to learn more about our unique products for #Alzheimers & #Parkinsons disease research. 🧠

Learn more 🍁 https://bit.ly/2II2Jeg

#AlphaSynuclein #Tau #AmyloidBeta #TDP43 #TTR #SOD1

Product Citation 🌟

Trichostatin A (TSA), commonly used in cancer studies, may also be #neuroprotective. In a neuronal model, TSA significantly improved cell viability in the presence of StressMarq's #AmyloidBeta 1-42 Oligomers.

Paper 🧩 https://bit.ly/4scYknu
Product 📚 https://bit.ly/4gYWkcj

StressMarq will be exhibiting at #ADPD2026 from March 17-21 in Copenhagen!📍

Visit us at Booth No. 70 to learn more about our unique products for #Alzheimers & #Parkinsons disease research. 🧠

Learn more 🍁 https://bit.ly/2II2Jeg

#AlphaSynuclein #Tau #AmyloidBeta #TDP43 #TTR #SOD1

StressMarq will be exhibiting at #ADPD2026 from March 17-21 in Copenhagen!📍

Visit us at Booth No. 70 to learn more about our unique products for #Alzheimers & #Parkinsons disease research. 🧠

Learn more 🍁 https://bit.ly/2II2Jeg

#AlphaSynuclein #Tau #AmyloidBeta #TDP43 #TTR #SOD1

Enhancing the Astrocytic Clearance of Amyloid Beta Astrocytic Sox9 promotes amyloid beta clearance and preserves cognition, revealing new astrocyte-driven therapeutic strategies for Alzheimer’s disease.

💫 Enhancing Sox9 in #astrocytes upregulates the MEGF10-mediated clearance of #AmyloidBeta plaques, safeguarding cognitive function in #neurodegenerative disease models.

Review the key findings from this study on the StressMarq blog ⭐ https://bit.ly/4qZ2Eq7

Product Citation ⏰

#Circadian dysfunction may increase #AD risk. Scientists utilized StressMarq's #AmyloidBeta PFFs to probe the role of brain border-associated #macrophages (BAMs) in enhancing the uptake of extracellular Aβ.

Paper 🔖 https://bit.ly/492YHKE
Product 🍎 https://bit.ly/3Mh0ekr

Product Citation 🎏

Border-associated #macrophages (BAMs) play a key role in the clearance of #AmyloidBeta. Scientists generated iPSC-derived BAM-like cells and tested their functionality using StressMarq’s Aβ 1-42 PFFs.

Paper 🎭 https://bit.ly/4sAlmWm
Aβ 1-42 🎋 https://bit.ly/3Mh0ekr

Multivalent nanomedicine targeting LRP1 at the blood–brain barrier accelerates amyloid-β clearance and restores cognitive function in Alzheimer’s models.
#AlzheimersDisease #AmyloidBeta #BloodBrainBarrier #Nanomedicine #Neurodegeneration
www.nature.com/articles/s41...

Amyloid β alters vascular CaV1.2 channel spatiotemporal properties Abstract figure legend Amyloid-β1-42 (Aβ1-42) triggers a male-specific signalling cascade influencing CaV1.2 spatiotemporal properties in cerebral vascular smooth muscle. The signalling pathway invol...

Just in time for the Holidays, our latest paper on how #amyloidbeta regulates the spatiotemporal properties of vascular L-type channels - implications for vascular function.

Great work by Jade and team! @ucdavis.bsky.social, @ucdavishealth.bsky.social

physoc.onlinelibrary.wiley.com/doi/10.1113/...

Experimental drug NU-9 halts Alzheimer’s disease in animal model before symptoms begin – BioNews Central The experimental drug NU-9 significantly reduced toxic amyloid beta oligomers and related neuronal damage in a mouse model of Alzheimer's disease, demonstrating potential as an early therapeutic intervention. ...

The #ExperimentalDrug NU-9 significantly reduced toxic #AmyloidBeta oligomers and related #NeuronalDamage in a mouse model of #AlzheimersDisease, demonstrating potential as an early #TherapeuticIntervention. 

BioRxiv Article

[BioRxiv] Conserved phenotype and function of human brain border-associated macrophages in iPSC-derived models

https://doi.org/10.64898/2025.12.11.693582

#neurodegeneration #biorxiv #amyloidbeta #amyloid

BioRxiv Article

[BioRxiv] The circadian clock regulates scavenging of fluid-borne substrates by brain border-associated macrophages

https://doi.org/10.64898/2025.12.08.693074

#neurodegeneration #biorxiv #amyloid #amyloidbeta #Alzheimers

BioRxiv Article

[BioRxiv] Age-specific Aβ-Tau interactions underlie spatial memory deficits in an APP/PS1 amyloidosis model

https://doi.org/10.64898/2025.12.06.689974

#neurodegeneration #biorxiv #amyloid #amyloidbeta #Alzheimers

BioRxiv Article

[BioRxiv] TREK1 upregulation is an endogenous mechanism delaying cognitive decline in Alzheimer's Disease

https://doi.org/10.1101/2025.10.16.682816

#neurodegeneration #biorxiv #amyloid #amyloidbeta #synapticdysfunction #Alzheimers

BioRxiv Article

[BioRxiv] Dual-action peptide shuttles rescue Cu-amyloid-beta-induced neurotoxicity and relocate Cu intracellularly

https://doi.org/10.1101/2024.09.04.611242

#neurodegeneration #biorxiv #amyloidbeta #neurotoxicity #amyloid

⚡Product Citation⚡

In #Alzheimers models generated with StressMarq’s #AmyloidBeta 1-42 Oligomers, scientists demonstrated that enhancing Sox9 in #astrocytes drives #phagocytosis of Aβ plaques and preserves #cognition.

Paper 🌟 https://bit.ly/48q5s7z
Aβ 1-42 Oligomers 🔎 https://bit.ly/4gYWkcj

Even modest amounts of physical activity may slow Alzheimer’s disease among at-risk older adults – BioNews Central Physical activity was associated with slower cognitive decline in older adults who had elevated levels of amyloid‑beta, a protein linked to Alzheimer’s disease. ...

#PhysicalActivity was associated with slower #CognitiveDecline in #OlderAdults who had elevated levels of #AmyloidBeta, a protein linked to #AlzheimersDisease.

Amyloid Beta Aggregation Protocol for Aβ Peptides | Hello Bio Discover our expert-designed Amyloid Beta protocol for in vitro and in vivo aggregation studies. Includes handling of Aβ monomers, tips to monitor aggregation, and fibril generation methods.

Amyloid Beta Aggregation Protocol #amyloidbeta #amyloid
➡️ hellobio.com/amyloid-beta...

6 Essential Protocols for Life Science Researchers Solid, reliable protocols are the key to turning tricky experiments into meaningful results. From Western blots to ELISAs, we’ve rounded up six essential step-by-step guides that scientists around the...

Looking for help with your Western blot? Need support with your ELISA assay? 🤔

Reliable protocols can turn tricky experiments into meaningful results & that's why we’ve put together 6 essential protocols for scientists: ow.ly/vWvT50X6XCb

#westernblot #elisaassay #amyloid #amyloidbeta #protocols

Product Citation: #AmyloidBeta 1-42 drives #Alzheimers disease pathology. Novel findings using StressMarq’s Amyloid Oligomers (A11) Antibody show that bovine #fibrinogen binds #Aβ42, suppressing fibril formation & reducing toxicity.

Paper 🌱 https://bit.ly/41MbX26
Product ✨ https://bit.ly/48HbvTw

I am very excited to let you know that my first PhD study has now been published in Nature Communications. 🧠📄 See the link to the article or read a summary in the post below! ⬇️

#alzheimersdisease #tau #amyloidbeta #neuroimaging #MRI #PET

Scientists discover the receptor that helps your brain clean itself—and fight Alzheimer’s Scientists at UCSF have uncovered how certain immune cells in the brain, called microglia, can effectively digest toxic amyloid beta plaques that cause Alzheimer’s. They identified a key receptor, ADGRG1,...

“Researchers at UC San Francisco identified a molecular #receptor that enables #microglia to gobble up and digest #AmyloidBeta plaques. Without the receptor, ADGRG1, the microglia barely nibbled on the toxic protein.”: buff.ly/Yj4Uqvs

via sciencedaily
#AlzheimersDisease #ClinicalResearch

Product Citation: A novel study suggests that protein misfolding may link ischemic injury to stroke-heart syndrome, contributing to #neurocardiac dysfunction. Findings were validated using StressMarq's #AmyloidBeta 1-42 Oligomers.

Paper 💬 https://bit.ly/4kKd5d0
Product⚡ https://bit.ly/4gYWkcj

🧠 Oligomeric #AlphaSynuclein, #Tau & #AmyloidBeta proteins have emerged as key players in #neurodegenerative diseases. Explore how StressMarq's high-quality oligomeric protein constructs, cited in top-tier research, are advancing pre-clinical drug discovery & disease models: https://bit.ly/4kE2Xn0

Advancements in Amyloid-Beta Research for Neurodegenerative Conditions — Nano Magazine The aggregation of Amyloid-beta (A-beta) proteins is a phenomenon intricately linked to neurodegenerative di...

#Nano #Technology #advancements #AmyloidBeta #conditions […]

[Original post on zephyrnet.com]

Optimizing Alpha Synuclein Seed Amplification Assays Seed amplification assays detect abnormal forms of alpha synuclein. An improved quiescent SAA method streamlines this process and maintains tissue integrity.

Using StressMarq's #BetaSynuclein, #GammaSynuclein & #AmyloidBeta 1-42 PFFs, scientists developed a quiescent seed amplification assay (QSAA) to detect #AlphaSynuclein aggregates in #cerebrospinal fluid & #skin samples.

Read about it on the blog! https://bit.ly/43QvnTN

StressMarq is pleased to announce that our #AlphaSynuclein, #Tau and #AmyloidBeta proteins for #neurodegenerative disease research are now available for purchase through the Science Exchange Marketplace.

For more information, please vist 🔬https://bit.ly/4kHRHpu

A Neuroprotective Compound in Traditional Chinese Medicine Thonningianin A (TA), a compound found in traditional Chinese medicine, exerts neuroprotective effects by binding to tau & amyloid beta proteins involved in AD.

StressMarq's #Tau (K18) P301L Mutant PFFs were employed to validate Thonningianin A, a plant-derived flavonoid, as an inhibitor of #Alzheimer's disease-related #AmyloidBeta & #Tau proteins.

Product details 💭 bit.ly/2mMPiSr
Read the blog 📬 bit.ly/43tgIib

Product Citation: #Synaptic dysfunction is an early result of #Alzheimer's disease. StressMarq's ENaC alpha Antibody was used to demonstrate the role of ENaC in modulating oligomeric #AmyloidBeta activity.

Paper 📕 bit.ly/4k4qUTK
Product 🧠 bit.ly/4cZUBTI